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What Tests Does My Poodle Need?

Health screening requirements differ by variety, and the differences are not intuitive. This is what each one actually needs, and what still cannot be tested at all.

Last reviewed July 17, 2026

Verified against OFA, July 2026.The CHIC requirements below were checked against OFA’s live breed listings. Be aware that the Poodle Club of America’s own site is currently inconsistent on one point: its health page still carries older wording giving the Standard three elective options, while its poodle information page lists the two cardiac examinations separately. OFA’s listing is the authority, and it gives the Standard four. Requirements do change, so confirm against ofa.org before making a breeding or registration decision.

CHIC requirements by variety

CHIC is a joint programme of OFA and the parent breed club. A dog earns a CHIC number when it has been screened according to the breed protocol and the results are publicly available in the OFA database. A CHIC number therefore records that testing was done and published. It is not a statement that a dog is healthy, and it is not a breeding recommendation.

Across all participating breeds, the dog must also be permanently identified by microchip or tattoo.

Chart comparing CHIC health testing requirements across Toy, Miniature, and Standard Poodles, showing which tests are required, which are elective, and which do not apply.
CHIC requirements side by side. The Standard is the only variety with an elective, choosing one of four.Save chart

Toy Poodle

  • REQUIREDPRA-prcd DNA testFrom an OFA-approved laboratory.
  • REQUIREDEye examination (ACVO)Yearly, by a board-certified veterinary ophthalmologist.
  • REQUIREDPatellar luxationOFA evaluation. Minimum age 12 months.

Miniature Poodle

  • REQUIREDPRA-prcd DNA testFrom an OFA-approved laboratory.
  • REQUIREDEye examination (ACVO)Yearly, by a board-certified veterinary ophthalmologist.
  • REQUIREDPatellar luxationOFA evaluation. Minimum age 12 months.
  • REQUIREDHip dysplasiaOFA or PennHIP evaluation.

Standard Poodle

  • REQUIREDHip dysplasiaOFA or PennHIP evaluation.
  • REQUIREDEye examination (ACVO)Yearly, by a board-certified veterinary ophthalmologist.

PLUS AT LEAST ONE OF:

  • Autoimmune thyroiditisOFA evaluation from an approved laboratory.
  • Sebaceous adenitisOFA evaluation by an approved dermatopathologist.
  • Cardiac, basicAuscultation by a licensed veterinarian.
  • Cardiac, advancedEchocardiography by a board-certified cardiologist.

The differences breeders most often miss

  • PRA-prcd DNA testing is a CHIC requirement for Toy and Miniature, but not for Standard.
  • Patellar luxation is required for Toy and Miniature, but not for Standard.
  • Hip evaluation is required for Miniature and Standard, but not for Toy.
  • The "choose at least one" elective structure exists only for the Standard, which picks one of four options.

A note on Moyen and Medium.CHIC recognizes three poodle varieties: Standard, Miniature, and Toy. There is no separate CHIC listing for Moyen or Medium. Some laboratories sell a “Moyen” profile, but that is a commercial category rather than a registry variety. Test according to the variety the dog is registered as.

DNA disease tests

These are the tests where a laboratory can read the genotype directly. A clear result here is meaningful and permanent, because a dog’s DNA does not change.

Progressive rod-cone degenerationPRA-prcd

Progressive retinal degeneration leading to blindness.

Varieties: Toy and Miniature (CHIC required); optional in StandardInheritance: Autosomal recessive
Rod-cone dysplasia 4 (late-onset PRA)PRA-rcd4

Late-onset retinal degeneration. Signs typically appear between five and twelve years, night and peripheral vision first.

Varieties: All three varietiesInheritance: Autosomal recessive
von Willebrand Disease Type 1vWD1

A bleeding disorder caused by deficient clotting factor.

Varieties: All; most emphasized in StandardsInheritance: Described inconsistently across laboratories

Some laboratories describe this as autosomal recessive, others as autosomal dominant with incomplete penetrance. Ask your laboratory how they interpret a carrier result before acting on it.

Neonatal Encephalopathy with SeizuresNEwS

Affected puppies are small and weak at birth and many die in the first week. Survivors develop ataxia, tremor, and seizures by four to six weeks.

Varieties: StandardInheritance: Autosomal recessive
Degenerative MyelopathyDM (SOD1)

Progressive degeneration of the spinal cord in adult dogs.

Varieties: All; commonly tested in StandardsInheritance: Autosomal recessive with incomplete penetrance

Incomplete penetrance means an at-risk result is not a diagnosis. Many at-risk dogs never develop clinical signs.

GM2 Gangliosidosis (Sandhoff disease)GM2

A fatal lysosomal storage disease. Onset around nine to twelve months with tremor and incoordination.

Varieties: ToyInheritance: Autosomal recessive
Osteochondrodysplasia (skeletal dwarfism)

Stunted growth visible from about three weeks, with splayed hind limbs, enlarged joints, and bent long bones.

Varieties: MiniatureInheritance: Autosomal recessive

Coat colour testing is a separate matter and is not a health test. If that is what you are looking for, start with the colour calculator or the merle reference centre.

Tests that apply to one variety only

Some poodle diseases are confined to a single variety. These are the ones most often missed, because a generic breed list lumps all three varieties together and a breeder reasonably assumes it applies to their dog.

Toy Poodle only

  • GM2 Gangliosidosis (Sandhoff disease)GM2

    A fatal lysosomal storage disease seen in the Toy. Onset around nine to twelve months.

Miniature Poodle only

  • Osteochondrodysplasia (skeletal dwarfism)SLC13A1

    Documented in the Miniature. The Poodle Club of America has reported that roughly one in ten Miniatures carries the variant, and that it is not confined to a few bloodlines.

Standard Poodle only

  • Neonatal Encephalopathy with SeizuresNEwS

    A Standard Poodle disease. Affected puppies are weak at birth, most die in the first week, and survivors seize by four to six weeks.

  • Day Blindness with Retinal DegenerationSPATA7

    A Standard Poodle disease identified at the University of Pennsylvania. Unlike day blindness in other breeds, it does not stay stable: affected dogs are blind in bright light from puppyhood, then degenerate to total blindness at around four to five years.

Shared rather than exclusive, but worth knowing: CDDY matters mainly in the Toy and Miniature, and PRA-prcd is a CHIC requirement for those two varieties but not for the Standard.

The extended panel, and how much to trust it

Beyond the CHIC requirements, laboratories offer a longer list of tests for poodles. Not all of them carry the same weight. Some are validated in poodles, some are on the panel because the allele turns up at low frequency, and some appear on generic breed lists with no poodle evidence behind them at all.

We have sorted them by how strong the poodle-specific evidence actually is, rather than listing them as though they were equivalent.

Chart sorting poodle DNA panel results into three bands: documented in poodles, on panels with limited evidence, and not validated in poodles.
Which panel results actually mean something for a poodle.Save chart
Documented in poodles

The variant is genuinely present in poodles with published evidence behind it. What a positive result means still depends on the variety and the condition, so read each entry rather than assuming a result is bad news.

Chondrodystrophy (CDDY)CDDY

In chondrodystrophic breeds such as the dachshund, this retrogene is associated with intervertebral disc disease. In poodles the picture is different, and a positive result is the norm rather than a warning sign.

Gene: FGF4 retrogene, chromosome 12Varieties: Common to near-fixed in Toy and Miniature; Standards largely clearInheritance: Dominant for leg length. The disc disease association is breed-dependent

Do not read a positive CDDY result in a poodle as a disc disease verdict. One copy is not established to raise IVDD risk within the breed, and in most cases two copies are not either. The variant is close to fixed in Miniatures, so nearly every dog would test positive. Treat it as one input to weigh alongside CDPA status, your veterinarian’s assessment, and the judgement of experienced breeders, not as a breeding disqualification on its own.

Day Blindness with Retinal DegenerationDB/RD

Affected dogs are blind in bright light from puppyhood, then undergo full retinal degeneration at around four to five years and lose vision in all lighting.

Gene: SPATA7 deletionVarieties: StandardInheritance: Autosomal recessive
On poodle panels, evidence limited

Included in commercial poodle panels, but the poodle-specific validation rests on allele frequency or extrapolation rather than confirmed poodle cases. Usually cheap as part of a panel; interpret a result cautiously.

Ehlers-Danlos Syndrome, "Poodle Type"

A connective tissue disorder producing fragile, hyperextensible skin.

Gene: TNXB, variants 1 and 2Varieties: Offered for Standard and MiniatureInheritance: Autosomal recessive, presenting as compound heterozygosity

The name is misleading. The dog in the original study was a mixed breed, not a poodle. One of the two alleles was found at low frequency in poodle and chihuahua populations, and the authors recommended monitoring. There is no published confirmed case in a poodle.

Congenital Methemoglobinemia

Blood carries oxygen poorly. Affected dogs have blue-tinged gums and tire easily, but are often otherwise well.

Gene: CYB5R3Varieties: Offered for Standard and MiniatureInheritance: Autosomal recessive

Marketed as a poodle type, but the definitive study sequenced pit bull terriers and pomeranians rather than poodles. Older clinical literature does list the Miniature Poodle.

Congenital Macrothrombocytopenia

Fewer but larger platelets. Clinically harmless in itself.

Gene: TUBB1Varieties: Offered for Standard and MiniatureInheritance: Autosomal recessive

Its real value is diagnostic rather than for breeding. Because it looks like a low platelet count, an affected dog can be misdiagnosed with immune-mediated thrombocytopenia and treated for a disease it does not have. Knowing the genotype prevents that.

Not validated in poodles

No published poodle frequency data and no confirmed poodle case. Some of these appear on generic breed lists; others arrive because a panel tests every dog for everything it offers. Either way, a result here is unlikely to tell you anything useful about a poodle, and none of them is a diagnosis.

MDR1 drug sensitivityABCB1

In affected breeds, causes severe reactions to certain common drugs including some parasiticides and sedatives.

Gene: ABCB1 nt230(del4)Varieties: None established in poodlesInheritance: Autosomal, incomplete dominance

A herding breed variant. Collies, Australian Shepherds, Shetland Sheepdogs and related breeds carry it. No poodle appears in the published breed frequency studies, and the major laboratories do not include it in their poodle panels.

ChondrodysplasiaCDPA

Produces the short-legged conformation of breeds such as the dachshund and corgi.

Gene: FGF4 retrogene, chromosome 18Varieties: None established in poodlesInheritance: Dominant

Where laboratories list it for poodles it sits under traits rather than diseases, and poodles are expected to test clear. Do not confuse it with CDDY above: they are different retrogenes on different chromosomes. CDPA status is, however, worth knowing when interpreting a CDDY result, which is why the two are best read together rather than in isolation.

Copper Toxicosis (accumulating and attenuating)ATP7B / ATP7A

In the breeds where it is established, copper builds up in the liver because the dog cannot excrete it efficiently, eventually causing liver damage.

Gene: ATP7B (accumulating), ATP7A and RETN (attenuating)Varieties: None established in poodlesInheritance: ATP7B incomplete dominance; ATP7A X-linked and complex

These are Labrador Retriever and Doberman Pinscher variants, sold by laboratories under names such as "Labrador Retriever Type." They arrive on a poodle report because the panel screens every dog for everything it offers, not because poodles were studied. A result carries little to no relevance for the breed and is not a diagnosis of liver disease.

ALT ActivityGPT

Associated with a baseline level of the liver enzyme ALT that sits at the low end of the normal range. It does not cause liver disease and produces no symptoms.

Gene: GPTVarieties: Not breed-specific, and not a poodle considerationInheritance: Codominant

This is not a health test at all, and Embark itself classifies it as a clinical tool rather than a health result. Its only practical use is that a dog with a naturally low baseline may not show an obviously raised ALT if its liver is later damaged, which is context your veterinarian can note. It is not diagnostic, it is not a clearance, and it is not a breeding consideration.

Hereditary CataractsHSF4

Early-onset cataract formation in the breeds where it is validated.

Gene: HSF4Varieties: None established in poodlesInheritance: Varies by breed

Established in the Staffordshire bull terrier, Boston terrier, and Australian shepherd. No poodle frequency data or case was found, and it is absent from the major poodle panels. Cataracts do occur in poodles, but this particular variant is not the reason, which is why the annual eye examination matters more here than a DNA test.

The bottom tier is included deliberately rather than left out. A breeder who reads a generic breed list will go looking for MDR1 or hereditary cataracts and wonder why we do not mention them. The answer is that the evidence does not support testing poodles for them, and that is more useful to know than silence.

Screening examinations

These are physical examinations rather than gene tests. Most expire, and several have a minimum age, which is the detail most often missed when a breeder plans a first litter.

Timeline chart showing the minimum age at which each poodle health test can be performed and how often it must be repeated.
Minimum ages and repeat intervals. DNA results never expire; most screening examinations do.Save chart
ExaminationPerformed byMinimum ageValid for
Eye examination (CAER)Annual re-examination is recommended. OFA notes this is a screening exam, not a comprehensive ocular evaluation.Board-certified ACVO ophthalmologistNot specified by OFA12 months from the date of exam
Hip dysplasiaCHIC accepts either an OFA radiographic evaluation or PennHIP and does not itself impose a 24-month minimum. An official OFA hip certification does require 24 months; below that a dog receives a preliminary evaluation, which is not a certification. OFA grades run Excellent, Good, and Fair (normal), then Borderline through Severe.OFA, or PennHIP as an accepted alternativeNo CHIC minimum; 24 months for OFA certificationPermanent once certified
Patellar luxationExamined awake; chemical restraint is not recommended. Evaluations under 12 months are consultations only.Attending veterinarian12 monthsPermanent
Cardiac, basicAny licensed veterinarian, by auscultation12 months12 months
Cardiac, advancedEchocardiography has been mandatory for the advanced database since October 2020.Board-certified veterinary cardiologist12 monthsCongenital clearance is permanent; adult-onset clearance is valid 12 months
Thyroid (autoimmune thyroiditis)Panel is free T4, cTSH, and thyroglobulin autoantibodies. OFA recommends testing at ages 2, 3, 4, 6, and 8.OFA-approved laboratory12 monthsRe-test periodically
Sebaceous adenitisRequires a minimum of two 6mm punch biopsies from the neck. Equivocal results should be re-tested in three to six months.OFA-approved dermatopathologist12 monthsRe-test every 1 to 2 years for breeding dogs

What has no DNA test

This section matters more than it looks. Several of the conditions poodle breeders worry about most cannot be tested for genetically at all. The Standard Poodle’s “choose at least one” elective structure exists largely because of this: the phenotypic screens are proxies, standing in for gene tests that do not exist.

Sebaceous adenitis

No DNA test exists. Diagnosis is by skin biopsy only, and the screening method can produce false negatives.

Addison's disease (hypoadrenocorticism)

No predictive DNA test. Genetic and environmental components are both involved, and research is ongoing. Monitored through blood work.

Bloat and gastric torsion (GDV)

No predictive DNA test.

Idiopathic epilepsy

A genetic predisposition is described, with onset typically between one and five years, but there is no validated DNA test.

The practical consequence: a screened dog is a dog that passed a screen on the day it was examined. That is genuinely valuable, and it is not the same thing as being clear. Anyone presenting a screening result as a permanent guarantee has misunderstood what the test does.

Sources

This page is an educational reference for breeders. It does not replace veterinary advice, laboratory consultation, or the current published requirements of OFA and the Poodle Club of America. Screening requirements change, and this page carries a review date so you can judge how current it is.